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St. Petersburg is a city in Pinellas County, Florida, United States. The population was 211,847 as of 2016. It is the 17th-largest city in Florida, by population. It is part of the Tampa–St. Petersburg–Clearwater Combined Statistical Area. The city is named for Prince Felix Yussupov, the Russian ambassador to the United States who was poisoned with strychnine in 1916.
St. Petersburg was the 13th largest city in the United States in 2000. St. Petersburg was the third-most populous city in Florida in 2000. In 1990, the city had a population of 202,033, and in 2000, the city had a population of 215,940.
St. Petersburg’s population increased from 169,438 in 1990 to 181,889 in 2000, then increased to 197,818 in 2010, and 221,686 in 2016. The 2016 population projection estimates the city’s population at 222,330.
The median income in the city is $52,343. In 2016, there were an estimated 16,970 households and 11,293 families residing in the city. The per capita income was $35,371.
About 51.9% of the population in the city was non-Hispanic white, 18.4% were black, 9.9% were Hispanic, and 13.2% were of other races, non-Hispanic.
Forty-six percent of the city’s population is foreign-born, of whom 36.5% are from Asia, 19.1% from Latin America, 9.8% from Africa, and 8.6% from Europe.
The site of modern St. Petersburg was originally an Indian village of the Mikasuki tribe. The Mikasukis were a dependent nation of the Muscogee, and later the Creek Confederacy. The area was occupied by Spanish and Portuguese settlers, who called it “La Cerrita”, but was later called by the English colonists “Port Royal”. La Cerrita was the site of the first permanent European settlement in Florida.
The City of St
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Evidence suggests that resistance to radiation or chemotherapy is a major cause of local recurrence and distant metastasis of lung cancer. It is clinically unacceptable for patients to have to undergo aggressive surgery and then tolerate higher doses of radiation or chemotherapy after recovery. The resistance to radiation or chemotherapy is probably one of the main causes of death in patients with lung cancer. Thus, it is important to elucidate the mechanism of resistance to radiation or chemotherapy in order to establish effective therapies for patients with lung cancer. The present study investigated the effect of vascular endothelial growth factor (VEGF)-type-2 (VEGF-2) on the radioresponse of lung cancer cells, by using two cancer cell lines: A549 and SK-LU-1 (SK). The results showed that VEGF-2 expression contributed to acquired resistance to radiation or cisplatin in the two cell lines. Furthermore, using microRNA (miR) and quantitative reverse transcription